Stress influences what we learn and remember. The hormone cortisol, which is released during stressful situations, can make emotional memories in particular stronger. But how exactly does cortisol help the brain build emotional memories?

In a new study, Yale researchers investigated just that. Specifically, they wanted to know how cortisol acts separately on brain circuits that track emotion and those that track memory. They found that cortisol not only helped people remember emotional experiences but also enhanced emotional memory by changing the dynamic brain networks associated with both memory and emotion.

“We all experience stress, and my lab is interested in understanding how stress can be helpful,” said corresponding author Elizabeth Goldfarb, an assistant professor of psychiatry at Yale School of Medicine and of psychology in the Faculty of Arts and Sciences.

The findings are published in the journal Science Advances.

In most narratives, the story of evolution is the story of organisms emerging from the ocean and eventually populating the land.

But for some species that evolution also involved a return trip. Dozens of major mammal and reptile groups ultimately made their way back to the beach and into the water. A new Yale study has undertaken the task of explaining when and how this happened — and which species fully re-committed to the life aquatic.

The study appears in the journal Current Biology.

SAN ANTONIO — April 14, 2026 — New research led by Southwest Research Institute (SwRI) integrated three types of machine learning models to generate solar magnetic patches with physical properties and used those as a query to find matching patches in real observations. This elevates generative artificial intelligence (AI) from a means to produce artificial data to a novel tool for scientific data interrogation, supporting applicability beyond the heliophysics domain.

Traumatic brain injury (TBI) affects over 69 million people yearly, and post‑TBI cognitive impairment (CTTI) often persists for years. The brain‑lymphatic axis – the central glymphatic system and peripheral lymphatic vessels – plays a key role. Glymphatic dysfunction (loss of AQP4 polarization) causes metabolic waste accumulation and neuroinflammation, while peripheral lymphatic stasis worsens cognitive decline. Most studies investigate the two systems in isolation; longitudinal data, non‑invasive biomarkers, and combined therapies are lacking. This review synthesizes current evidence, identifies gaps, and proposes integrated axis‑targeted approaches.