Tubulin prevents toxic protein clumps in the brain, fighting back neurodegeneration
Peer-Reviewed Publication
Updates every hour. Last Updated: 2-Jun-2026 21:15 ET (3-Jun-2026 01:15 GMT/UTC)
Researchers at Baylor College of Medicine have discovered a potential new strategy to fight back against Alzheimer’s and Parkinson’s diseases, conditions that are linked to the toxic accumulation of Tau and alpha synuclein protein clumps in the brain. The team reports that tubulin, the building block of microtubules, the cell’s internal ‘railway tracks,’ can stop Tau and alpha synuclein from forming toxic clumps and instead steer them into their normal, healthy roles. The findings support redefining tubulin, from a passive casualty of disease to an active protector against toxic protein aggregation.
A machine-learning model developed by Weill Cornell Medicine investigators may provide clinicians with an early warning of a complication that can occur late in pregnancy.
State laws that ban insurance prior authorization for buprenorphine—a leading medication for opioid use disorder—may not help more patients stay in treatment for the recommended minimum of 180 days, Weill Cornell Medicine researchers report. Though prescription buprenorphine can be a life-saving treatment that relieves opioid cravings and withdrawal symptoms, adherence to the medication is low.
Phage Therapy and the Medicine of Tomorrow: Scientists Meet in Valencia to Define the Path to Clinical Integration
Valencia, Spain. June 9–10, 2026
Researchers, clinicians, and biotechnology innovators from around the world will gather in Valencia, Spain, for Targeting Phage Therapy 2026, an international scientific meeting dedicated to advancing bacteriophage based strategies to combat antimicrobial resistance and difficult to treat bacterial infections.
GLP-1 drugs have come to be thought of as “wonder drugs,” yet the mechanisms behind their many new health benefits have remained unclear. New research from the Salk Institute reveals the mechanism behind one of these benefits: promoting pancreatic beta cells’ health and stress tolerance, long-term. The findings show how GLP-1 drugs can trigger broad genomic responses.