Research finds soil inorganic carbon rise offsets organic carbon loss, masking ecosystem degradation beneath stable total carbon pools.

A landmark lipidomic study reveals that ceramides modulate salicylic and jasmonic acid pathways to govern resistance to Sporisorium scitamineum.

The security of digital signatures has been a focus of research in emerging area. Threshold signature is a technique designed for distributed scenarios, allowing the signing key to be divided into multiple shares. This approach can resist single point failures of device and ensure key security. However, most existing surveys on threshold signature schemes are either not comprehensive or cover only a limited range of signature types.

Sub-headline: BIT researchers introduce Malcom to tackle cross-domain encrypted traffic detection using self-supervised learning.

Deep neural networks (DNNs) are demonstrated to be vulnerable to adversarial examples. Adversarial training is mainstrem method to improve adversarial robustness of DNNs, which augments the training set with adversarial examples and adopts adversarial regularization loss to improve the robustness of DNNs. Existing adversarial training methods are facing the challenge to balance the accuracy and robustness.

Sub-headline: Beihang University researchers introduce a multi-authority ABE scheme, tackling dynamic access control challenges in cloud data.

This cover review identifies CCN1 as a central molecular hub that integrates sensitive skin and skin aging. Upregulated by ultraviolet exposure, oxidative stress, or intrinsic aging, CCN1 drives barrier dysfunction, chronic inflammation, extracellular matrix degradation, and pigmentation abnormalities. In sensitive skin, acute CCN1 overactivation amplifies inflammation and increases permeability, while in aging skin, its sustained overexpression promotes inflammaging, collagen loss, and fibroblast senescence. Acting through integrin-dependent pathways such as NF‑κB, MAPK, and TGF‑β, CCN1 remodels the dermal‑epidermal microenvironment. Targeting CCN1 or its downstream signaling offers a promising strategy to restore skin homeostasis, shifting the focus from symptomatic relief toward key‑node regulation.