Cobalt-free LiNiO₂ (LNO) is considered a promising cathode for its high energy density and cost-effectiveness. However, its structural instability under deep delithiation severely limits practical application in next-generation lithium-ion batteries (LIBs). Microstructure engineering enhances structural stability through precisely controlled lattice modulation strategies, particularly via high-valence element doping which effectively stabilizes the crystal framework through strong bonding characteristics and charge compensation effects.

Flexible electronics is profoundly leading the wave of transformation in fields such as wearable devices, health monitoring, and intelligent robots, and material innovation is undoubtedly the core driving force behind this revolution. As a new type of material prepared by compounding liquid metals (LM) with other materials, LM thin films, with their unique properties, have become an ideal candidate in the field of flexible electronics preparation, laying a solid foundation for the vigorous development of flexible electronics technology.

Los Angeles, CA – March 9, 2026 – The Terasaki Institute for Biomedical Innovation (TIBI) and Keck Graduate Institute (KGI) have announced a new collaborative research partnership designed to accelerate biomedical innovation through joint research programs, faculty collaboration, and expanded student training opportunities.

The NF-κB-inducing kinase (NIK), a molecule pivotal for immune system development and function, shows significant yet complex potential as a therapeutic target. The comprehensive review, published by Professor Shao-Cong Sun’s team, systematically details the expanding understanding of NIK. This article summarizes its canonical roles, its newly discovered functions independent of NF-κB, and its pathological contributions to autoimmunity, framing both the opportunities and challenges in targeting this critical immune regulator.

Rotator cuff tears often heal with stiff, dysfunctional scar tissue, limiting recovery. A new study reveals why tendon regeneration fails after injury. Using single-cell profiling of tens of thousands of cells from patient tendon samples, the study maps the first atlas of human tendon scarring and identifies pro-fibrotic stem cells, senescent tendon cells, scar-forming macrophages, and transitioning endothelial cells. Targeting key fibrotic signals reduced scarring in animal models, suggesting new therapeutic strategies.