Hebrew SeniorLife’s Right Care, Right Place, Right Time (R3) has a new name — Health Allies.

Health Allies brings healthy aging teams into affordable senior housing communities to help older adults navigate the health care system, optimize their health and well-being, remain independent longer, and enjoy their best possible lives. It is currently in place at 18 affordable housing communities across Greater Boston.

Nuclear medicine is playing an increasingly important role in the diagnosis and treatment of Alzheimer’s disease, helping clinicians visualise the biological changes associated with the condition and guide decisions about new therapies. Amyloid PET imaging is being used to confirm the presence of amyloid pathology and support treatment decisions for patients receiving anti-amyloid drugs, while tau PET scans can provide additional information about disease progression and prognosis. At the 39th Annual Congress of the European Association of Nuclear Medicine (EANM’26), taking place from 17 to 21 October in Vienna, experts will discuss first experiences with these PET scans in routine clinical use and explore the latest advances in nuclear medicine.

Collagen plays a critical role in keeping heart valves strong, but the heart is surrounded by enzymes designed to break down collagen. So how can heart valves function efficiently decade after decade? A new study shows that the biomechanical tension applied to these valves every time your heart beats plays a key role in protecting the collagen from breaking down.

Experts from the University of Nottingham have discovered how a common genetic mutation disrupts the positioning of a key protein during cell division, revealing a new mechanism behind acute myeloid leukaemia and a potential avenue for future treatment.

Acute myeloid leukaemia (AML) is an aggressive blood cancer that disrupts the production of healthy blood cells. Around one-third of patients carry a mutation in a gene called NPM1, which causes its protein to accumulate in the wrong part of the cell and plays a key role in driving the disease.

Until now, research has largely focused on how the mutated protein moves between the cell's nucleus and the surrounding cytoplasm. But a new study, led by Dr Daniel Booth and Dr Claire Seedhouse at the University of Nottingham's School of Medicine, reveals that what happens during cell division itself is also critical.