Sharks, spines and speed: The hidden architecture behind different swimming styles
Peer-Reviewed Publication
Updates every hour. Last Updated: 24-Oct-2026 22:16 ET (25-Oct-2026 02:16 GMT/UTC)
What gives sharks their extraordinary speed, agility and endurance? It’s not just their muscles or powerful tails – it’s the hidden architecture inside their spines. Using high-resolution micro-CT imaging, researchers found that great white, shortfin mako and porbeagle sharks have rigid, mineral-rich spines built for speed, while sand tiger sharks evolved more flexible vertebrae for maneuverability. Common thresher sharks have reinforced spinal structures for powerful tail strikes, and basking sharks possess lightly mineralized spines adapted for slow, energy-efficient cruising.
Technion researchers have identified a previously unknown mechanism that enables bacteria to develop antibiotic resistance far more rapidly than previously understood. The study, led by Dr. Idan Yelin and Prof. Roy Kishony of the Technion’s Faculty of Biology and published in Nature Microbiology, reveals how bacteria can quickly produce dozens of copies of genes that confer resistance.
Using AmpliFinder, a new computational tool, the researchers analyzed more than 10,000 laboratory-evolved bacterial samples and discovered that non-canonical gene amplifications are both common and highly efficient. Experiments in Escherichia coli and Acinetobacter baumannii showed that this mechanism enables bacteria to adapt to progressively higher concentrations of antibiotics. Disrupting the process could offer a new way to slow the emergence of resistance and improve the effectiveness of antibiotic treatments.
Researchers at IBEC have discovered that epithelial tissues adapt to prolonged stretching by slowly reorganising their keratin cytoskeleton into large multicellular networks. Using advanced live imaging, engineered tissue models and computational simulations, the team found that this process eventually pushes the cell nucleus out of its surrounding keratin cage. The findings reveal a previously unknown mechanism by which tissues respond to sustained mechanical stress, with potential implications for development, organ physiology and diseases linked to defective keratin organisation.