Researchers at the ALS Center Netherlands at University Medical Center Utrecht have identified new genetic abnormalities that play a role in the development of ALS. As a result, the proportion of patients for whom a genetic cause can be identified of the neuromuscular disease has increased from 20 to 25 percent. The discovery also improves understanding of the disease and may, in time, support the development of targeted treatments for a subset of patients.

Colour coding on food product labels is becoming more common. How does it influence consumers and their dietary choices? Recent research by scientists from SWPS University, the University of Wisconsin, and the University of Massachusetts indicates that colour coding is much more effective than simple nutritional tables, and it is all due to the way the brain responds to benefit and risk signals.

Lifestyle and dietary changes may help people living with endometriosis.

Early recovery after stroke is dominated by powerful biological repair processes, and increasing the therapy dose over and above standard care very early after stroke may not enhance those processes.

A nationwide clinical RCT trial led by Dr. Kazumi Kimura at Kumamoto University Hospital has found that adding catheter ablation to standard anticoagulant therapy did not significantly reduce the risk of recurrent stroke or major cardiovascular events in patients with atrial fibrillation who had recently experienced a stroke.

The connection between testosterone and well-being is weaker than many people think. Although there are clear health connections, a higher testosterone level is not always the key to well-being, according to a thesis at the University of Gothenburg.

Understanding how proteins interact with their own mirror images enabled a Kobe University research team to design a small mirror protein that disables a causal factor of Alzheimer’s disease, amyloid-beta.

For more than two thousand patients who had cycled through years of antidepressant regimens without relief, the problem was never solely in their heads. A new study published in Brain Medicine tracked 2,197 individuals across six years and found that specific dysfunctions of the parasympathetic and sympathetic nervous systems, namely alpha-sympathetic withdrawal and parasympathetic excess, were starving the brain of adequate blood flow in ways that mimicked or magnified depressive states. Once clinicians identified and corrected those autonomic imbalances using low-dose pharmacologic and lifestyle interventions, 95 percent of subjects experienced symptom relief, plummeting from an average of 23.2 reported symptoms at baseline to 5.2 at final follow-up. The findings challenge the assumption that patients who fail standard antidepressants are simply treatment resistant.