Recently, a research team from Jilin University and Hainan University reported a jointed experimental and theoretical study on Coulomb focusing in energy-resolved angular streaking. It was found that photoelectrons emitted at different initial ionization times can converge to the same emission angle due to previously unexpected Coulomb focusing in nonadiabatic tunneling. The findings have significant implications for decoding quantum tunneling dynamics under the classically forbidden barrier with attosecond angular streaking.

A new pregnancy health cohort study, led by BGI Genomics in collaboration with medical centers from 12 cities in China, has identified common complications, high-risk groups, and geographical trends through biological samples collected across China.

In a paper published in MedComm-Oncology, an international team of scientists present evidence of fossilized cell nuclei and chromosomes within preserved cartilage in a baby duck-billed dinosaur. This dinosaur belongs to Hypacrosaurus and comes from a nesting ground discovered in 1988 by paleontologist Jack Horner in Late Cretaceous sediments of Northwest Montana.

Ischemic injury causes dynamic damage to the native extracellular matrix (ECM), which plays a key role in tissue homeostasis and regeneration by providing structural support, facilitating force transmission, and transducing key signals to cells. The main approach aimed at repairing injury to ischemic tissues is restoration of vascular function. Due to their potential to form capillary niches, endothelial cells (ECs) are of greatest interest for vascular regeneration. Integrin binding to ECM is crucial for cell anchorage to the surrounding matrix, spreading, migration, and further activation of intracellular signaling pathways. In this study, we proposed to establish an in-situ engineering strategy to remodel the ECM at the ischemic site to guide EC endogenous binding and establish effective EC/ECM interactions to promote revascularization. We designed and constructed a dual-function molecule (LXW7)₂-SILY, which is comprised of two functional domains: the first one (LXW7) binds to integrin αvβ3 expressed on ECs, and the second one (SILY) binds to collagen. In vitro, we confirmed (LXW7)₂-SILY improved EC adhesion and survival. After in situ injection, (LXW7)₂-SILY showed stable retention at the injured area and promoted revascularization, blood perfusion, and tissue regeneration in a mouse hindlimb ischemia model.