COVID-19 linked to increased asthma risk – vaccine offers protection
Peer-Reviewed Publication
Updates every hour. Last Updated: 11-Oct-2025 20:10 ET (12-Oct-2025 00:10 GMT/UTC)
People who have had COVID-19 are at increased risk of developing certain inflammatory diseases of the airways, such as asthma, hay fever and chronic sinusitis. However, vaccination against the SARS-CoV-2 virus appears to reduce the risk, according to a comprehensive epidemiological study led by researchers at Karolinska Institutet.
As the nation gears up for the rollout of an updated COVID-19 vaccine, a new study shows the economic benefits of continued broad vaccination in adults. In fact, the country would ultimately save more money that it would spend on vaccinating every person over age 65 with a single dose of an updated mRNA vaccine against coronavirus, the study concludes.
Public health officials had an unprecedented tool for near-instant, widespread communication during the COVID-19 pandemic and mpox epidemic: social media.
Now, one of the first studies of its kind, led by a health policy expert with the Texas A&M University School of Public Health, has found that X (Twitter at the time of the events) brought advantages — as well as disadvantages — in getting the word out.
Key Findings:
AI-powered Aging Clock: The new proteomic clock predicts biological age with high accuracy (R²=0.84, MAE=2.68 years) and captures accelerated aging signatures in severe lung disease cases—patients with severe COVID-19 (and likely fibrosis) exhibited biological ages nearly three years older than healthy controls.
Distinct Molecular Signatures: Analysis with the ipf-P3GPT generative model revealed both shared and unique gene expression patterns between aging lungs and fibrotic disease, highlighting that IPF is not just accelerated aging but entails unique pathological processes.
Pathway-Level Insights: The study identified four key pathways (TGF-ß signaling, oxidative stress, inflammation, ECM remodeling) as central to both IPF and aging, but involved differently at the gene level.
Groundbreaking research led by a Swansea University academic has revealed a synthetic glycosystem — a sugar-coated polymer nanoparticle — that can block Covid-19 from infecting human cells, reducing infection rates by nearly 99%.
Researchers from the Translational Genomics Research Institute (TGen), part of City of Hope, and the Lundquist Institute for Biomedical Innovation at Harbor-UCLA Medical Center have identified a potential biomarker for long COVID.
If the findings of their study are confirmed by other research centers, the biomarker could be the first specific and quantifiable indicator for confirming long COVID. Currently, clinicians confer a diagnosis of long COVID based upon a collection of symptoms that patients develop after SARS-CoV-2 infection.
The first paper from a multi-year clinical research study has been published in The Lancet Infectious Diseases: Dynamics of Endemic Virus Re-emergence in Children in the USA Following the COVID-19 Pandemic (2022-2023): A Longitudinal Immunoepidemiologic Surveillance Study and demonstrates how the approach can improve modeling to better predict future outbreaks.
The paper shares findings from a multicenter clinical research study, one of many studies that are part of the recently launched PREMISE (Pandemic Response Repository through Microbial and Immune Surveillance and Epidemiology) program, led by Dr. Daniel Douek at the National Institutes of Health’s (NIH) Vaccine Research Center (VRC). Data collected during the first year of the PREMISE study, 2022-2023, shows for the first time how non-pharmaceutical interventions such as masking and distancing targeted towards SARS-CoV-2 during the pandemic also decreased circulation rates of and population immunity to common respiratory pathogens in children. The study provides new evidence-based insight into what was driving the large post-pandemic rebound in these diseases and enables more accurate predictions for the future.