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The capsid of the virus-derived retrotransposon Copia, a parasitic genome element, mediates synaptic plasticity at the Drosophila neuromuscular junction

Peer-Reviewed Publication

PLOS

The capsid of the virus-derived retrotransposon Copia, a parasitic genome element, mediates synaptic plasticity at the Drosophila neuromuscular junction

image: 

A reduction of the retrotransposon at the Drosophila NMJ leads to a drastic increase in synaptogenesis and plasticity (-copia). The structure of Copia (center), shows a large retroviral like structure with pores (right panel), that are very similar to those found in HIV-1 capsids, these pores and like structures are needed for reverse transcription of viral genomes.  Altogether we found that the retroviral nature of Copia regulates a physiological function. 

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Credit: M’Angale PG et al., 2025, PLOS Biology, CC-BY 4.0 (https://creativecommons.org/licenses/by/4.0/)

In your coverage, please use this URL to provide access to the freely available paper in PLOS Biology: https://plos.io/42Ly2Pr  

Article title: Capsid transfer of the retrotransposon Copia controls structural synaptic plasticity in Drosophila

Author countries: United States

Funding: This work was supported by NIH Grant R01NS112492 to TT. The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript.


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