Researchers proposed an image-aided terminal guidance attitude control method for Explosively Formed Penetrator (EFP)-equipped multirotors to achieve long-range precision strikes, solving disturbance, EFP dispersion and visual-attitude coupling problems. Published in Defence Technology, the work built a multi-source EFP dispersion model, designed an NTSM-STO composite controller, and validated its fast response, strong robustness and high hit rate via Monte Carlo simulations and real flight tests.

This review synthesizes the evolution of aquatic ecosystem health assessment and proposes a paradigm shift from descriptive monitoring to predictive, intelligent governance. Led by Yong Liu from Peking University, the research team introduces a gene-to-landscape framework that integrates multi-omics, environmental DNA, ecological observation, and AI-enabled remote sensing to enable early warning, causal attribution, and anticipatory decision support for freshwater ecosystems under global change.

This review summarizes flexible electromagnetic induction-type tactile sensors that convert touch-induced magnetic-flux variation into electrical signals. It establishes an integrated framework linking magnets, coils, soft materials, signal processing and circuits, and discusses their relevance to wearable electronics, medical monitoring, human-machine interfaces and intelligent robotics.

Researchers tracked longitudinal alterations in adipose and muscle tissue through routine CT scans. Worsening visceral fat tissue degeneration predicts complications for compensated chronic hepatitis B-related cirrhosis, while sustained muscle loss signals risk in decompensated patients.

Ulinastatin (UTI), a protein naturally found in human urine, exerts hepatoprotective effects by attenuating inflammation, oxidative stress, and cell death, and modulating immunity and hemostasis. This review synthesizes existing evidence on UTI's therapeutic potential across seven liver conditions, from sepsis-induced hepatic injury to liver cancer.

Immune checkpoints (ICs) are receptors that regulate immune responses through both activating and inhibitory mechanisms. Inflammatory microenvironment modulates IC expression through cytokines, metabolites, and oxidative signals. Immune checkpoint inhibitors not only enhance antitumor immunity but also reshape systemic immune networks. This review summarizes the molecular and cellular mechanisms by which ICs regulate inflammatory responses and highlights their roles across diverse pathological conditions.