Proposed model of the mechanism of PpSnRK1α-mediated phosphorylation of PpNAC6/PpNAC36 in N-regulated γ-decalactone biosynthesis in peach fruit. (IMAGE)

Nanjing Agricultural University The Academy of Science

Proposed model of the mechanism of PpSnRK1α-mediated phosphorylation of PpNAC6/PpNAC36 in N-regulated γ-decalactone biosynthesis in peach fruit.

Caption

Proposed model of the mechanism of PpSnRK1α-mediated phosphorylation of PpNAC6/PpNAC36 in N-regulated γ-decalactone biosynthesis in peach fruit. Under low-N conditions, PpNAC6 and PpNAC36 maintain high expression levels, promoting the transcription of γ-decalactone biosynthesis genes and thereby enhancing γ-decalactone production. Following excessive N addition, PpSnRK1α, which responds to high-N levels, is induced and expressed. PpSnRK1α interacts with  PpNAC6 and PpNAC36, phosphorylating PpNAC36 but not PpNAC6. Additionally, under high-N conditions, the cellular localization of PpNAC6 and PpNAC36 is no longer restricted to the nucleus. Consequently, the transcriptional activation of γ-decalactone biosynthesis genes by PpNAC6 and PpNAC36 is suppressed, leading to a reduction in γ-decalactone production.

Credit

Horticulture Research

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